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Joined 3 years ago
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Cake day: July 2nd, 2023

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  • I get what you’re saying but it seems overly complicated and unnecessary risk. We know UV exposure is not good, so why do it? Yes the body can make vitamin D with some exposure, but what’s your opposition to simply taking a supplement and eating vitamin D rich foods or fortified drinks?

    Your setup needs to account for timing, distance, and duration appropriately without any easy feedback to determine those parameters are correct. Even then there are still risks including eye exposure as you said.

    What you’re saying can be done, but it’s complicated and not without risk when there are easier and safer alternatives. Personally, I take a medication that increases my risks from sun exposure, so I always wear sunscreen and avoid direct sunlight when possible. I’ve found taking a supplement to be super easy and it’s generally a good idea to take a high quality, well rounded supplement since it can account for other nutrients your diet may be lacking at any point in time (ex. Was my diet lower in iron or some vitamin this week? No big deal since I supplement anyways)






  • Oh look, bullshit information without a source. Also correlation is not the same as causation. Hopefully this snippet is short enough for you to ingest:

    https://www.nature.com/articles/d41586-025-02876-1

    The study led by Ahlqvist harnessed data from nearly 2.5 million children born in Sweden between 1995 and 2019 and — from the country’s extensive health records — data on acetaminophen prescriptions during pregnancy and on self-reported use collected by midwives, as well as whether children later received autism diagnoses.

    The study showed that around 1.42% of children exposed to acetaminophen during pregnancy were autistic, compared with 1.33% of children who were not exposed ─ a “very small” difference, says Ahlqvist.

    The team also compared pairs of siblings (born to the same person), in which one had been exposed to acetaminophen and one had not. Siblings share half of their genome, a similar upbringing and maternal health, so differences in autism are more likely to be due to the drug. Using this method, the researchers found no association between acetaminophen and autism — which supports the idea that links found in other studies could be explained by confounding factors.

    A large, high-quality study from Japan of more than 200,000 children — also using sibling comparisons and published this year — found no link between acetaminophen use in pregnancy and autism.